EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors

White, Robert E., Raemer, Patrick C., Naresh, Kikkeri N., Meixlsperger, Sonja, Pinaud, Laurie, Rooney, Cliona, Savoldo, Barbara, Coutinho, Rita, Boedoer, Csaba, Gribben, John, Ibrahim, Hazem A., Bower, Mark, Nourse, Jamie P., Gandhi, Maher K., Middeldorp, Jaap, Cader, Fathima Z., Murray, Paul, Muenz, Christian and Allday, Martin J. (2012) EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors. Journal of Clinical Investigation, 122 4: 1487-1502. doi:10.1172/JCI58092

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Author White, Robert E.
Raemer, Patrick C.
Naresh, Kikkeri N.
Meixlsperger, Sonja
Pinaud, Laurie
Rooney, Cliona
Savoldo, Barbara
Coutinho, Rita
Boedoer, Csaba
Gribben, John
Ibrahim, Hazem A.
Bower, Mark
Nourse, Jamie P.
Gandhi, Maher K.
Middeldorp, Jaap
Cader, Fathima Z.
Murray, Paul
Muenz, Christian
Allday, Martin J.
Title EBNA3B-deficient EBV promotes B cell lymphomagenesis in humanized mice and is found in human tumors
Journal name Journal of Clinical Investigation   Check publisher's open access policy
ISSN 0021-9738
Publication date 2012-04-01
Year available 2012
Sub-type Article (original research)
DOI 10.1172/JCI58092
Open Access Status Not Open Access
Volume 122
Issue 4
Start page 1487
End page 1502
Total pages 16
Place of publication Ann Arbor, MI, U.S.A.
Publisher American Society for Clinical Investigation
Language eng
Keyword Epstein-Barr-Virus
T-Lymphocytes
Immune-System
Growth Transformation
Burkitts-Lymphoma
In-Vitro
Gene
Responses
Expression
Infection
Q-Index Code C1
Q-Index Status Confirmed Code
Grant ID 077489
R01CA108609
10/02
KFS-02652-08-2010
11-0516
310030_126995
Institutional Status UQ

Document type: Journal Article
Sub-type: Article (original research)
Collections: Official 2013 Collection
School of Medicine Publications
 
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Citation counts: TR Web of Science Citation Count  Cited 60 times in Thomson Reuters Web of Science Article | Citations
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Created: Tue, 31 Jul 2012, 09:52:08 EST by Matthew Lamb on behalf of School of Medicine