Dietary advanced glycation end products as an environmental contributor to type 1 diabetes

Borg, Danielle J. and Forbes, Josephine M. (2013) Dietary advanced glycation end products as an environmental contributor to type 1 diabetes. IMARS Highlights, 8 5: 21-22.

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Name Description MIMEType Size Downloads
Author Borg, Danielle J.
Forbes, Josephine M.
Title Dietary advanced glycation end products as an environmental contributor to type 1 diabetes
Journal name IMARS Highlights
Publication date 2013-09-18
Sub-type Letter to editor, brief commentary or brief communication
Open Access Status
Volume 8
Issue 5
Start page 21
End page 22
Total pages 2
Place of publication Cleveland, OH, USA
Publisher International Maillard Reaction Society
Collection year 2014
Language eng
Formatted abstract
Type 1 diabetes (T1D) is defined by the autoimmune destruction of the insulin-producing beta cells, within the pancreatic islets of Langerhans. The growing prevalence of T1D in the last three decades suggests a change in environmental stimuli rather than variations in genetic susceptibility [1]. Several putative nongenetic factors resulting in likely risk for T1D have been suggested, including increased maternal age at delivery, maternal enteroviral infections and caesarean section. Recently, focus has been on early dietary changes as a potential environmental contributor to T1D [2]. We are particularly interested in advanced glycation end products as a causative trigger for T1D...
Q-Index Code CX
Q-Index Status Provisional Code
Institutional Status Non-UQ

Document type: Journal Article
Sub-type: Letter to editor, brief commentary or brief communication
Collections: Non HERDC
School of Medicine Publications
 
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Created: Thu, 24 Apr 2014, 14:55:42 EST by Dominique Rossouw on behalf of Mater Clinical School