"White" Nevi and "Red" Melanomas: Association with the RHC Phenotype of the MC1R Gene

Zalaudek, I., Meiklejohn, W., Argenzanio, G., Thurber, A. E. and Sturm, R. A. (2009) "White" Nevi and "Red" Melanomas: Association with the RHC Phenotype of the MC1R Gene. The Journal of Investigative Dermatology, 129 5: 1305-1307. doi:10.1038/jid.2008.378


Author Zalaudek, I.
Meiklejohn, W.
Argenzanio, G.
Thurber, A. E.
Sturm, R. A.
Title "White" Nevi and "Red" Melanomas: Association with the RHC Phenotype of the MC1R Gene
Journal name The Journal of Investigative Dermatology   Check publisher's open access policy
ISSN 0022-202X
Publication date 2009-05-04
Year available 2008
Sub-type Letter to editor, brief commentary or brief communication
DOI 10.1038/jid.2008.378
Open Access Status
Volume 129
Issue 5
Start page 1305
End page 1307
Total pages 3
Editor P. R. Bergstresser
Place of publication New York
Publisher Elsevier Science
Collection year 2009
Language eng
Subject 060103 Cell Development, Proliferation and Death
060403 Developmental Genetics (incl. Sex Determination)
920117 Skin and Related Disorders
920102 Cancer and Related Disorders
C1
Abstract In 2002, we reported on three patients presenting with melanocytic nevi lacking pigmentation, which we named "white" dyplastic melanocytic nevi (DMN) due to their peculiar clinical appearance for white to pale red macules with accentuated skin markings and a silvery "shining" when observed with tangential light (Zalaudek et al.,2002). Notably, all three patients had melanoma, and in one patient white DMN were associated with two primary amelanotic melanomas (AMMs).
Q-Index Code C1
Q-Index Status Confirmed Code

Document type: Journal Article
Sub-type: Letter to editor, brief commentary or brief communication
Collections: 2009 Higher Education Research Data Collection
Institute of Modern Languages - Publications
 
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Citation counts: TR Web of Science Citation Count  Cited 13 times in Thomson Reuters Web of Science Article | Citations
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Created: Mon, 16 Mar 2009, 11:56:05 EST by Cody Mudgway on behalf of Institute for Molecular Bioscience