Double Trouble for Type 1 Angiotensin Receptors in Atherosclerosis

Thomas, Walter G (2005) Double Trouble for Type 1 Angiotensin Receptors in Atherosclerosis. New England Journal of Medicine, 352 5: 506-508. doi:10.1056/NEJMcibr043928

Author Thomas, Walter G
Title Double Trouble for Type 1 Angiotensin Receptors in Atherosclerosis
Journal name New England Journal of Medicine   Check publisher's open access policy
ISSN 0028-4793
Publication date 2005-02-03
Sub-type Article (original research)
DOI 10.1056/NEJMcibr043928
Volume 352
Issue 5
Start page 506
End page 508
Total pages 3
Place of publication Boston, MA, United States
Publisher Massachusetts Medical Society
Language eng
Subject 060110 Receptors and Membrane Biology
060111 Signal Transduction
060199 Biochemistry and Cell Biology not elsewhere classified
060108 Protein Trafficking
060602 Animal Physiology - Cell
110201 Cardiology (incl. Cardiovascular Diseases)
110903 Central Nervous System
Abstract The researchers then found that the levels and activity of activated factor XIII (factor XIIIa) transglutaminase were higher in the monocytes from hypertensive patients than in those from normotensive subjects. Transglutaminases are a family of calcium-dependent enzymes that catalyze the cross-linking of proteins, including membrane proteins, through the amino acids glutamine and lysine.5 Factor XIIIa is best known for cross-linking the fibrin network during blood coagulation.
Keyword Proteins
blood pressure
Ace Inhibitors
Q-Index Code C1
Q-Index Status Confirmed Code
Institutional Status Non-UQ

Document type: Journal Article
Sub-type: Article (original research)
Collections: Excellence in Research Australia (ERA) - Collection
School of Biomedical Sciences Publications
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Citation counts: TR Web of Science Citation Count  Cited 4 times in Thomson Reuters Web of Science Article | Citations
Scopus Citation Count Cited 7 times in Scopus Article | Citations
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